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What does P2-12 stand for?

P2-12 stands for postnatal 2- to 12-day-old

This definition appears rarely

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Given that ageing-related degenerative changes are universal in the musculoskeletal system18, 19, 20, we investigated the impact of ageing on murine muscle-derived stem/progenitor cells (MDSPCs). MDSPCs are multipotent cells isolated from postnatal skeletal muscle through an established preplate technique21, 22. They have a capacity for long-term proliferation, are resistant to oxidative and inflammatory stress, show multilineage differentiation and self-renew, induce neovascularization, and stimulate regeneration of bone, skeletal and cardiac muscles21, 23, 24, 25.
w. caused reversible blockage of beta waves in the electroretinogram in immature mice and rats, indicating retinotoxicity (Potts et al. , 1960). The timing of treatment of mice was quite critical. After 10-11 days postnatal it was difficult to produce significant lesions of the retina (Olney, 1969a). A study of the glutamate metabolizing enzymes of the retina of glutamate-treated rats indicated a decrease in glutaminase activity, an increase in glutamic aspartate transaminase, and no change in glutamyl synthetase and glutaminotransferase.
MRI has been widely used to investigate the asphyxiated infant2, 3, 6, 9, 19, 34–36. The abnormalities identified on imaging will vary according to the magnet and sequences used and the postnatal age of the infant at the time of the scan. The pattern of injury seen on MRI is related to the type and severity of the insult. Severe acute asphyxia is associated with lesions in the basal ganglia, thalami, brain stem, hippocampus and the corticospinal tracts around the central fissure3, 23, 27, 28, 30, 31.
dosed orally with 10% monosodium glutamate (2 gm/kg), showed characteristic brain lesions (Geil, 1970). Monosodium glutamate caused reversible blockage of beta wave in the electroretinogram in immature mice and rats indicating retinotoxicity (Potts et al. , 1960). The timing of treatment of mice was quite critical. After 10 to 11 days postnatal.
Glucose is an essential source of energy during embryonic growth and fetal development and is vital for mesenchymal cell differentiation, chondrogenesis, and skeletal morphogenesis. Glucose is an important metabolic fuel for differentiated chondrocytes during postnatal.