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What does EAD stand for?

EAD stands for early after-depolarizations


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This can also explain why hypokalemia is associated with an increased likelihood for producing Torsade de pointes, since it will increase drug effects to the point of producing early after depolarizations (EADs). This interaction can be reproduced in both isolated Purkinje fibers and transmural myocardial preparations in vitro (Roden & Hoffman, 1985, Antzelevitch & Burashnikov, 2011).
{Pressure x Radius} / {2 x wall thickness} measured during diastole C) Wall tension 21. {Pressure x Radius} / {2 x wall thickness} D) Compliance measured at any point in time E) Stiffness 22. Change in pressure / Change in volume 23. Change in volume / Change in pressure **************************************************************** EKG/Arrhythmia questions 24. Rhythms with a heart rate < 60/min are known as: A) Triggered early after depolarizations.
This form of long QT syndrome most likely involves mutations of the 'human ether-a-go-go related gene' (hERG) on chromosome 7. The hERG gene (also known as KCNH2) is part of the rapid component of the potassium rectifying current (IKr). (The IKr current is mainly responsible for the termination of the cardiac action potential, and therefore the length of the QT interval. ) The normally functioning hERG gene allows protection against early after depolarizations.