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k. a. MRL/lpr) differs from the congenic (control) MRL/+ strain by a defect in membrane apoptotic-signaling Fas protein, which is due to a retrotransposon in the Fas gene [50, 51]. In addition to the typical signs of peripheral SLE, including autoantibodies, skin disease, arthritis, lymphadenopathy, and nephritis, MRL/lpr mice develop a constellation of behavioral outcomes referred to as “autoimmunity-associated behavioral syndrome.