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CD11b–/loLy6Chi OCPs expand in SKG arthritis and have T cell–suppressive activity in vitro and immunomodulatory activity in vivo, yet are not sufficient to prevent the development of zymosan-induced SKG arthritis. We thus asked whether OCPs derived from arthritic SKG mice differ in their suppressive activity from BALB/c mice. We found that CD11b–/loLy6Chi OCPs isolated from arthritic SKG mice are equivalent to wild-type BALB/c mice in their in vitro suppressive activity (Figure 7F).
Despite the inherent limitations of all animal models, several rodent models have significantly progressed our understanding of the fundamental mechanisms underpinning rheumatoid arthritis and contributed to several current major advances in treatment. These models include the induced arthritis models such as collagen-induced arthritis, collagen-antibody-induced arthritis, zymosan-induced arthritis.